Additional file 6 of Neural defects caused by total and Wnt1-Cre mediated ablation of p120ctn in mice

Additional file 6: Figure S6. Organizational abnormality of β-catenin expression in the neural folds and NTD of 18–22 somite p120fl/fl;Wnt1Cre mutant mice. Immunofluorescence of p120ctn (a, d) and β-catenin (b, e; overlay in c, f) in the cranial neural tube of a control 18–22 somite embryo (a-c), or in the cranial neural folds/NTD of a mutant 18–22 somite embryo with NTD (d-f). In the control embryo, β-catenin was nicely coexpressed with p120ctn. In the mutant embryo, p120ctn was lost in the NTD (d), but β-catenin was still expressed, although focally with aggregated appearance (e, arrows). Scale bar: 10 μm.