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Additional file 11 of Multi-omics integration identifies key upstream regulators of pathomechanisms in hypertrophic cardiomyopathy due to truncating MYBPC3 mutations

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posted on 2021-03-25, 08:38 authored by J. Pei, M. Schuldt, E. Nagyova, Z. Gu, S. el Bouhaddani, L. Yiangou, M. Jansen, J. J. A. Calis, L. M. Dorsch, C. Snijders Blok, N. A. M. van den Dungen, N. Lansu, B. J. Boukens, I. R. Efimov, M. Michels, M. C. Verhaar, R. de Weger, A. Vink, F. G. van Steenbeek, A. F. Baas, R. P. Davis, H. W. Uh, D. W. D. Kuster, C. Cheng, M. Mokry, J. van der Velden, F. W. Asselbergs, M. Harakalova
Additional file 11: Table S3 (A) Differentially expressed genes between HCM and control hearts from the RNA-seq data. (B) Enriched GO terms and pathways by up-regulated genes in HCM versus control hearts. (C) Enriched GO terms and pathways by down-regulated genes in HCM versus control hearts

Funding

The Netherlands Foundation for Cardiovascular Excellence NWO VENI grant NWO VIDI grant ZonMW-NWO VICI grant Erasmus MC fellowship grant RM fellowship grant of the UMC Utrecht Wilhelmina Children’s Hospital research funding Netherlands Cardiovascular Research Initiative: An initiative with the support of the Dutch Heart Foundation UCL Hospitals NIHR Biomedical Research Centre H2020 European Research Council

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    Clinical Epigenetics

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    Keywords

    HCMMYBPC3Histone acetylomeTranscriptomeProteomeTranscription factors

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