MOESM9 of p66ShcA functions as a contextual promoter of breast cancer metastasis
journal contributionposted on 16.01.2020 by Kyle Lewis, Alex Kiepas, Jesse Hudson, Julien Senecal, Jacqueline Ha, Elena Voorand, Matthew Annis, Valerie Sabourin, Ryuhjin Ahn, Rachel Selva, Sébastien Tabariès, Brian Hsu, Matthew Siegel, Matthew Dankner, Eduardo Canedo, Mathieu Lajoie, Ian Watson, Claire Brown, Peter Siegel, Josie Ursini-Siegel
Any type of content formally published in an academic journal, usually following a peer-review process.
Additional file 9: Figure S9. p66ShcA does not induce oxidative stress in primary tumors in a model of triple negative breast cancer. Quantification of the percentage of 4-HNE positive pixels in 4T1-537 parental, p66-CR, p66-CR (WT) and p66-CR (S36A) mammary tumors. The data is shown as positivity ± SEM and is representative of 9–10 tumors per group. Representative images are shown below each graph.