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Additional file 2: of The role of podocyte damage in the etiology of ischemia-reperfusion acute kidney injury and post-injury fibrosis

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posted on 2019-03-28, 05:00 authored by Yi Chen, Liyu Lin, Xuan Tao, Yankun Song, Jiong Cui, Jianxin Wan
Changes in urinary protein, BUN and Scr in each group from D0 to D7 (−x ± s, n = 10) There is no significant difference of urine protein, Scr and BUN in AKI mouse model from day 0 to day 7. (DOCX 20 kb)

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The Young Talents Training Program of the Fujian Provincial Health System of China

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    BMC Nephrology

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    Keywords

    PodocyteAcute kidney injury (AKI)Chronic kidney disease (CKD)SynaptopodinNephrinCD2APTRPC6

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