Additional file 1 of Senolytic compounds control a distinct fate of androgen receptor agonist- and antagonist-induced cellular senescent LNCaP prostate cancer cells

Additional file 1: Fig. S1. Enzalutamide treatment significantly induces CDKN2A (p16INK4a) expression. Fig. S2. GT, ABT263, and MK2206 inhibit LNCaP cells proliferation through apoptosis induction in a concentration dependent manner. Fig. S3. p-RIP3 is dephosphorylated by alkaline phosphatase (FAST AP). Fig. S4. GT enhances detachment of cells after androgen-induced cellular senescence. Fig. S5. ABT263 and MK2206 induce LNCaP cell detachment. Fig. S6. Androgen regulates Bcl-2 family members transcription levels.