Additional file 11: of Differential roles of STAT1 and STAT2 in the sensitivity of JAK2V617F- vs. BCR-ABL-positive cells to interferon alpha

Schubert, Claudia; Allhoff, Manuel; Tillmann, Stefan; MaiĂŠ, Tiago; Costa, Ivan; Lipka, Daniel; Schemionek, Mirle; Feldberg, Kristina; Baumeister, Julian; BrĂźmmendorf, Tim; Chatain, Nicolas; Koschmieder, Steffen

doi:10.6084/m9.figshare.7943792.v1

13045_2019_722_MOESM11_ESM.pdf (155.08 kB)

Additional file 11: of Differential roles of STAT1 and STAT2 in the sensitivity of JAK2V617F- vs. BCR-ABL-positive cells to interferon alpha

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posted on 2019-04-02, 05:00 authored by Claudia Schubert, Manuel Allhoff, Stefan Tillmann, Tiago MaiĂŠ, Ivan Costa, Daniel Lipka, Mirle Schemionek, Kristina Feldberg, Julian Baumeister, Tim BrĂźmmendorf, Nicolas Chatain, Steffen Koschmieder

Figure S10. H3K9 acetylation profile at ISGs. The ChIP-seq acetylation was visualized with the Integrative Genomics Viewer (IGV). Shown are the H3K9 acetylation peaks at the genomic regions of Irf1, Irf17, Irf9, and Stat1 in 32D-JAK2V7F (JAK2V617F) (red), 32D-BCR-ABL (blue), and 32D-EV (green). (PDF 108 kb)

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IFNa BCR-ABL JAK2V617F MPN Polycythemia vera Chronic myeloid leukemia STAT factors ISG CRISPR/Cas9

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Additional file 11: of Differential roles of STAT1 and STAT2 in the sensitivity of JAK2V617F- vs. BCR-ABL-positive cells to interferon alpha

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