%0 Journal Article %A Müller, Michaela %A Jacobi, Eric %A Sakimura, Kenji %A Malinow, Roberto %A von Engelhardt, Jakob %D 2018 %T Additional file 5: of NMDA receptors mediate synaptic depression, but not spine loss in the dentate gyrus of adult amyloid Beta (Aβ) overexpressing mice %U https://springernature.figshare.com/articles/journal_contribution/Additional_file_5_of_NMDA_receptors_mediate_synaptic_depression_but_not_spine_loss_in_the_dentate_gyrus_of_adult_amyloid_Beta_A_overexpressing_mice/7245629 %R 10.6084/m9.figshare.7245629.v1 %2 https://springernature.figshare.com/ndownloader/files/13345664 %K NMDA receptor %K Amyloid Beta %K Alzheimer’s disease %K GluN2B %K GluN2A %X S5. CT100(I716F) overexpression does not influence total dendritic length in adult mice. a Examples of traced DG granule cells of the GluN2Afl/fl mouse line. b The number of intersections analyzed by Sholl analysis is not changed by CT100(I716F) overexpression, GluN2A subunit deletion and GluN2A deletion in combination with CT100(I716F) overexpression. Mean ± SEM. Total dendritic length is not different between the groups. c Examples of traced DG granule cells of the GluN2Bfl/fl mouse line. d Sholl analysis of the number of intersections shows subtle changes in dendritic complexity in GluN2B−/−/CT100(I716F) cells compared to their respective control (GluN2B−/−). Mean ± SEM. Total dendritic length is not different between the groups. Bar graphs show median ± IQR.; dendr. = dendritic, morph. = morphology (PDF 133 kb) %I figshare