Additional file 8: of Integrated biology approach reveals molecular and pathological interactions among Alzheimer’s Aβ42, Tau, TREM2, and TYROBP in Drosophila models Michiko Sekiya Minghui Wang Naoki Fujisaki Yasufumi Sakakibara Xiuming Quan Michelle Ehrlich Philip De Jager David Bennett Eric Schadt Sam Gandy Kanae Ando Bin Zhang Koichi Iijima 10.6084/m9.figshare.6067103.v1 https://springernature.figshare.com/articles/dataset/Additional_file_8_of_Integrated_biology_approach_reveals_molecular_and_pathological_interactions_among_Alzheimer_s_A_42_Tau_TREM2_and_TYROBP_in_Drosophila_models/6067103 Table S7. Functional enrichment of DEGs identified in Tau/TREM2WT/TYROBP, Tau/TREM2R47H/TYROBP files. (XLSX 23 kb) 2018-03-29 05:00:00 Alzheimer’s disease Amyloid-β (Aβ) peptides Microtubule-associated protein tau TYROBP (tyrosine kinase binding protein) TREM2 (triggering receptor expressed on myeloid cells 2) Differential expression Gene co-expression network Gene module Synaptophagy Immune function Neurodegeneration