Additional file 8: of Integrated biology approach reveals molecular and pathological interactions among Alzheimer’s Aβ42, Tau, TREM2, and TYROBP in Drosophila models
Michiko Sekiya
Minghui Wang
Naoki Fujisaki
Yasufumi Sakakibara
Xiuming Quan
Michelle Ehrlich
Philip De Jager
David Bennett
Eric Schadt
Sam Gandy
Kanae Ando
Bin Zhang
Koichi Iijima
10.6084/m9.figshare.6067103.v1
https://springernature.figshare.com/articles/dataset/Additional_file_8_of_Integrated_biology_approach_reveals_molecular_and_pathological_interactions_among_Alzheimer_s_A_42_Tau_TREM2_and_TYROBP_in_Drosophila_models/6067103
Table S7. Functional enrichment of DEGs identified in Tau/TREM2WT/TYROBP, Tau/TREM2R47H/TYROBP files. (XLSX 23 kb)
2018-03-29 05:00:00
Alzheimer’s disease
Amyloid-β (Aβ) peptides
Microtubule-associated protein tau
TYROBP (tyrosine kinase binding protein)
TREM2 (triggering receptor expressed on myeloid cells 2)
Differential expression
Gene co-expression network
Gene module
Synaptophagy
Immune function
Neurodegeneration