10.6084/m9.figshare.c.3842200_D3.v1 Sylvia Torres-Odio Sylvia Torres-Odio Jana Key Jana Key Hans-Hermann Hoepken Hans-Hermann Hoepken Júlia Canet-Pons Júlia Canet-Pons Lucie Valek Lucie Valek Bastian Roller Bastian Roller Michael Walter Michael Walter Blas Morales-Gordo Blas Morales-Gordo David Meierhofer David Meierhofer Patrick Harter Patrick Harter Michel Mittelbronn Michel Mittelbronn Irmgard Tegeder Irmgard Tegeder Suzana Gispert Suzana Gispert Georg Auburger Georg Auburger Additional file 1: Table S1. of Progression of pathology in PINK1-deficient mouse brain from splicing via ubiquitination, ER stress, and mitophagy changes to neuroinflammation Springer Nature 2017 Parkinson’s disease Ubiquitin kinase PINK1 Mitochondrial dysfunction Antiviral response Neuroinflammation 2017-08-02 05:00:00 Dataset https://springernature.figshare.com/articles/dataset/Additional_file_1_Table_S1_of_Progression_of_pathology_in_PINK1-deficient_mouse_brain_from_splicing_via_ubiquitination_ER_stress_and_mitophagy_changes_to_neuroinflammation/5271247 Global transcriptome profile of Pink1 −/− brain regions (C = cerebellum, M = midbrain, S = striatum) across the mouse lifespan (6 W = 6 weeks, 24 W = 24 weeks = 6 months, 18 M = 18 months), showing all expression dysregulations which were consistent in all regions at all ages with significance (multiple testing adjusted p value <0.05). ID identifies the Affymetrix microarray chip probe. AveExpr quantifies the average expression strength among the tissues and ages for each transcript. F, P value, and AdjPvalue refer to statistical values. For each column, 3 mutant versus 3 WT tissues were analyzed to represent a specific brain region and age. (XLS 27 kb)