Additional file 1: of Exogenous hydrogen sulfide restores cardioprotection of ischemic post-conditioning via inhibition of mPTP opening in the aging cardiomyocytes
Hongzhu Li
Chao Zhang
Weiming Sun
Lina Li
Bo Wu
Shuzhi Bai
Hongxia Li
Xin Zhong
Rui Wang
Lingyun Wu
Changqing Xu
10.6084/m9.figshare.c.3618515_D1.v1
https://springernature.figshare.com/articles/journal_contribution/Additional_file_1_of_Exogenous_hydrogen_sulfide_restores_cardioprotection_of_ischemic_post-conditioning_via_inhibition_of_mPTP_opening_in_the_aging_cardiomyocytes/4386020
Figure S1. The change of the cell viability and apoptosis in the normal primary cultured neonatal cardiomyocytes. Figure S2. The change of caspase-3, caspase-9 and Bcl-2 mRNA levels in the normal primary cultured neonatal cardiomyocytes. Figure S3. The effect of exogenous H2S on p-ERK1/2 and p-GSK-3β mRNA levels in the d-galactose age-induced cardiomyocytes. Figure S4. The effect of exogenous H2S on p-PI3K, p-Akt and p-GSK-3β mRNA levels in the d-galactose age-induced cardiomyocytes. Figure S5. The effect of exogenous H2S on PKC-ξ mRNA levels in the cell membrane in the d-galactose age-induced cardiomyocytes.
2015-07-30 05:00:00
Hydrogen sulfide
Post-conditioning
Mitochondrial permeability transition pore
Aging cardiomyocytes