Additional file 1: of Exogenous hydrogen sulfide restores cardioprotection of ischemic post-conditioning via inhibition of mPTP opening in the aging cardiomyocytes Hongzhu Li Chao Zhang Weiming Sun Lina Li Bo Wu Shuzhi Bai Hongxia Li Xin Zhong Rui Wang Lingyun Wu Changqing Xu 10.6084/m9.figshare.c.3618515_D1.v1 https://springernature.figshare.com/articles/journal_contribution/Additional_file_1_of_Exogenous_hydrogen_sulfide_restores_cardioprotection_of_ischemic_post-conditioning_via_inhibition_of_mPTP_opening_in_the_aging_cardiomyocytes/4386020 Figure S1. The change of the cell viability and apoptosis in the normal primary cultured neonatal cardiomyocytes. Figure S2. The change of caspase-3, caspase-9 and Bcl-2 mRNA levels in the normal primary cultured neonatal cardiomyocytes. Figure S3. The effect of exogenous H2S on p-ERK1/2 and p-GSK-3β mRNA levels in the d-galactose age-induced cardiomyocytes. Figure S4. The effect of exogenous H2S on p-PI3K, p-Akt and p-GSK-3β mRNA levels in the d-galactose age-induced cardiomyocytes. Figure S5. The effect of exogenous H2S on PKC-ξ mRNA levels in the cell membrane in the d-galactose age-induced cardiomyocytes. 2015-07-30 05:00:00 Hydrogen sulfide Post-conditioning Mitochondrial permeability transition pore Aging cardiomyocytes