%0 Journal Article %A Xue, Haowei %A Lu, Jinsen %A Yuan, Renxiang %A Liu, Jinli %A Liu, Yehai %A Wu, Kaile %A Wu, Jing %A Du, Juan %A Shen, Bing %D 2016 %T MOESM1 of Knockdown of CLIC4 enhances ATP-induced HN4 cell apoptosis through mitochondrial and endoplasmic reticulum pathways %U https://springernature.figshare.com/articles/journal_contribution/MOESM1_of_Knockdown_of_CLIC4_enhances_ATP-induced_HN4_cell_apoptosis_through_mitochondrial_and_endoplasmic_reticulum_pathways/4360982 %R 10.6084/m9.figshare.c.3610610_D1.v1 %2 https://springernature.figshare.com/ndownloader/files/7087076 %K Chloride intracellular channel 4 %K Head and neck squamous carcinoma %K Apoptosis %K Mitochondrial membrane potential %K Endoplasmic reticulum stress %K Adenosine triphosphate %X Additional file 1: Figure S1. Effect of CLIC4 or scrambled siRNA on CLIC4 expression. Effect of CLIC4 or scrambled siRNA on CLIC4 expression. Summarized data showing the expression level of CLIC4. HN4 cells were transfected with CLIC4 or scrambled siRNA, and then were treated with (ATP-Con, ATP-CLIC4) or without (Con, Con-CLIC4) 100 μmol/L ATP for 3 h. β-Tubulin was used as a loading control. Values are shown as the mean ± SE. n = 3. *P < 0.05. vs. the control (Con) group,# P < 0.05 vs. the ATP control (ATP-Con) group. %I figshare