Additional file 1: Figure S1. of Aspirin inhibits epithelial-to-mesenchymal transition and migration of oncogenic K-ras-expressing non-small cell lung carcinoma cells by down-regulating E-cadherin repressor Slug Poulami Khan Argha Manna Shilpi Saha Suchismita Mohanty Shravanti Mukherjee Minakshi Mazumdar Deblina Guha Tanya Das 10.6084/m9.figshare.c.3608963_D1.v1 https://springernature.figshare.com/articles/figure/Additional_file_1_Figure_S1_of_Aspirin_inhibits_epithelial-to-mesenchymal_transition_and_migration_of_oncogenic_K-ras-expressing_non-small_cell_lung_carcinoma_cells_by_down-regulating_E-cadherin_repressor_Slug/4356425 p53 mutation exerts increased migratory effect in combination with oncogenic K-ras-expressing system on NSCLC cells migration. Phase contrast images (left panels) depicting migration of NCI-H522 cells (oncogenic K-ras/mutant p53), mutant p53-reconstituted H1299 cells (wild type K-ras/mutated p53), A549 cells (oncogenic K-ras/wild type p53), and wild type p53 reconstituted H1299 cells (wild type K-ras/wild type p53). Graphical representation of percent cell migration in wound healing assay (right panel) with inset showing immunoblot analysis for the transfection efficiency of p53 - R175H clone in H1299 cells and p53 - cDNA in H1299 cells. Scale bar: 100 μm. (TIFF 2457 kb) 2016-01-26 05:00:00 Aspirin E-cadherin Migration NSCLCs Oncogenic K-ras p65NFκB